Summary of ssbd-repos-000203

SSBD:database
URL

Name
ssbd-repos-000203 (203-Tanabe-SingleDyn)
URL
DOI
-

Title
Images of signaling molecules dynamic in Dictyostelium discoideum cells.
Description
-
Submited Date
-
Release Date
2022-03-31
Updated Date
-
License
Funding information
-
File formats
Data size
254.5 MB

Organism
Dictyostelium discoideum
Strain
gcΔ cell, Dictyostelium discoideum AX2 (NCBI:txid366501)
Cell Line
-
Genes
gc
Proteins
LimE, PHPKB, PIP3, sGC, sGCN, sGCWT

GO Molecular Function (MF)
cAMP binding
GO Biological Process (BP)
NA, chemotaxis
GO Cellular Component (CC)
NA
Study Type
Chemotaxis, Eukaryota, Eukaryota/metabolism, Animals, Signal Transduction, Chemotaxis/physiology
Imaging Methods
confocal microscopy

Method Summary
-
Related paper(s)

Yuki Tanabe, Yoichiro Kamimura, Masahiro Ueda (2018) Parallel signaling pathways regulate excitable dynamics differently to mediate pseudopod formation during eukaryotic chemotaxis., Journal of cell science, Volume 131, Number 23

Published in 2018 Dec 5 (Electronic publication in Dec. 5, 2018, midnight )

(Abstract) In eukaryotic chemotaxis, parallel signaling pathways regulate the spatiotemporal pseudopod dynamics at the leading edge of a motile cell through the characteristic dynamics of an excitable system; however, differences in the excitability and the physiological roles of individual pathways remain to be elucidated. Here, we found that two different pathways, mediated by soluble guanylyl cyclase (sGC) and phosphoinositide 3-kinase (PI3K), caused similar all-or-none responses for sGC localization and phosphatidylinositol 3,4,5-trisphosphate production but with different refractory periods, by undertaking simultaneous observations of the excitable properties of the two pathways in Dictyostelium cells. Owing to the shorter refractory period, sGC signaling responded more frequently to chemoattractants, leading to pseudopod formation with higher frequency. sGC excitability was regulated negatively by its product cGMP and by cGMP-binding protein C (GbpC) through the suppression of F-actin polymerization, providing the underlying delayed negative-feedback mechanism for the cyclical pseudopod formation. These results suggest that parallel pathways respond to environmental cues on different timescales in order to mediate chemotactic motility in a manner based on their intrinsic excitability.
(MeSH Terms)

Contact(s)
Masahiro Ueda
Organization(s)
Osaka University , Graduate School of Frontier Biosciences , Laboratory for Cell Signaling Dynamics
Image Data Contributors
Quantitative Data Contributors

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